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[求助]我的兒子吃線~已看獸醫~請問有可以不開刀的方法嗎?
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發表在 貓咪保健,
時間 2006-03-10 06:31 PM,
作者 changyuan
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下面引用由B可女夭在 2006/03/09 10:49am 發表的內容：
1. 品種 男
3. 年齡 Yes
5. 預防注射 : Yes 預防針種類 / 品牌 :先靈五合一
6. 就醫前主訴 晚上嘔吐~但他以前有時會因為吃太快~把剛吃下去的吐出來~
...
基本上你的獸醫在作法上並沒有特殊令人可議之處.
配合他的診察看看, 後續再回來跟大家說.
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我同學家剛出生的貓咪沒肛門
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發表在 貓咪保健,
時間 2006-03-08 04:37 PM,
作者 changyuan
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下面引用由yukilululala在 2006/03/07 09:33pm 發表的內容：
怪叔叔 救救我吧
要我念符咒隔空救貓嗎?
已經有貓友建議你了...趕快去吧!!
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[求助]觸診血檢全部正常卻不吃東西....
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發表在 貓咪保健,
時間 2006-03-08 04:24 PM,
作者 changyuan
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下面引用由雙貓夫人在 2006/03/08 10:01am 發表的內容：
品種 公/結紮
年齡 尿道容易結晶, 目前吃HILL'S C/D處方飼料
生化報告:
CRSC 0.8 MG/DL
...
就這幾種檢查項目看不出問題在哪裡.
請醫生幫它做更多項的檢查, 包括CBC, 總蛋白, 白蛋白...等.
如果可以的話, 空腹12小時後照X光看看腹腔, 也可能是毛球症或之類的問題 .
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腎上腺素皮質機能亢進？
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發表在 貓咪保健,
時間 2006-03-08 04:17 PM,
作者 changyuan
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沒力氣翻譯. 先嘗試讀讀看, 有疑問再說.
從內科聖經摘錄下來的內容可知真的有人在胡說八道.
我氣是在氣這個!!
HYPERADRENOCORTICISM IN CATS
Hyperadrenocorticism is an endocrine disorder not commonly recognized in cats. Over a 10-year period, one university clinic diagnosed Cushing's syndrome in more than 800 dogs and in only 34 cats.26
Similar to the incidence seen in dogs, 80% to 85% of cats have PDH, and 10% to 15% have an FAT. Adenomas and carcinomas occur with equal frequency. Adrenal tumors may secrete excessive amounts of steroids other than cortisol. Two recent case reports described cats with clinical signs of hyperadrenocorticism caused by a progesterone-secreting adrenal mass.87,88
Clinical Findings
Middle-aged to older cats are most commonly affected with hyperadrenocorticism. There does not seem to be a breed predilection; however, approximately 70% of feline cases are female. The most common clinical signs are polydipsia, polyuria, and polyphagia. Most authors have assumed that these symptoms are caused by diabetes mellitus, which develops in approximately 80% of cases, and that they therefore are late signs of hyperadrenocorticism. However, a recent case study described polydipsia and polyuria in two cats with hyperadrenocorticism that did not have concurrent diabetes mellitus and in one cat that had hyperadrenocorticism for 8 months before diabetes mellitus developed.89 Other frequent findings include pendulous abdomen, generalized muscle wasting, lethargy, and obesity. Many cats have dermatologic symptoms, including hair loss; unkempt haircoat; truncal or patchy alopecia; fragile, thin skin prone to traumatically induced tears (so-called feline fragile skin syndrome); and secondary infections (including demodicosis). Hepatomegaly and weight loss occur less frequently.
Hyperglycemia is the most frequent laboratory abnormality. Only 10% of dogs with hyperadrenocorticism develop overt diabetes mellitus, whereas as many as 80% of cats with Cushing's syndrome are diabetic. Cats seem to be more sensitive to the diabetogenic effects of steroids than dogs, and in many cases it is only after diabetes mellitus has been diagnosed that hyperadrenocorticism is suspected. Insulin resistance is a typical feature of diabetes mellitus caused by hyperadrenocorticism. However, it should be noted that not all cats with Cushing's syndrome and concurrent diabetes mellitus are insulin resistant.
page 1610
page 1611
Other findings include hypercholesterolemia and an increase in ALT activity, which may be caused by hyperadrenocorticism or diabetes mellitus. Some cats have elevated ALP activity. Because the activity of a steroid-induced isoenzyme is not increased, this elevation is thought to be associated with diabetes mellitus (and hepatic lipidosis) rather than with hyperadrenocorticism.90
Specific Endocrine Testing
A diagnosis of hyperadrenocorticism should be based on the results of one or more of the screening tests (urine C:C ratio, ACTH stimulation test, LDDS test). The endogenous ACTH concentration, an HDDS test, and adrenal ultrasonography can be used to differentiate PDH from an FAT in cats.
Urine Creatinine to Cortisol Ratio
Slightly more than 70% of cortisol (free cortisol and metabolites) is eliminated in the urine in dogs, whereas only 18% is eliminated in the urine of cats. Despite this difference, the urine C:C ratio of cats with hyperadrenocorticism is significantly higher than that of healthy cats, and this test can be used in the diagnosis of Cushing's syndrome.91 Urine should be collected by the owner at home. It is important to remember that false-positive results may be caused by other disease processes. Goosens et al.91 reported a reference range of 2 to 36 × 10-6 based on the results of 42 healthy cats. Our reference range, using 31 healthy cats, was 0 to 4 × 10-6. This discrepancy is possibly due to the different types of radioimmunoassay used. The amount of cortisol metabolites measured varies among assays, therefore it is critical that reference ranges be established for each assay.
ACTH Stimulation Test
In both dogs and cats, the ACTH stimulation test is mainly a test of adrenal reserve, and its major role is to rule in or rule out hypoadrenocorticism. The test's sensitivity is low, and because a variety of nonadrenal illnesses can cause abnormal test results, its specificity is also low. According to the test protocol most often used, blood samples for cortisol determination are taken before and 30 and 60 minutes after intramuscular administration of 125 μg/cat of a synthetic polypeptide containing the first 24 aminoacids of ACTH (e.g., Cortrosyn, Synacthen). The time to maximum peak cortisol response is longer and the maximum cortisol concentration is significantly higher after intravenous administration than after intramuscular administration. Therefore the protocol for intravenous administration is to collect blood samples before and 60 and 90 minutes after administration of ACTH. Because peak effect of cortisol is less consistent in the cat than in the dog, two post-ACTH samples are recommended. Reference ranges for the cat are slightly lower than those for the dog and should be established for the laboratory and protocol used. In our laboratory, the upper limit for the normal post-ACTH cortisol concentration using the intramuscular protocol is 13 μg/dL.92 We consider levels between 13 and 16 μg/dL to be borderline, and a concentration above 16 μg/dL to be consistent with hyperadrenocorticism.
Low-Dose Dexamethasone Suppression Test
The degree and duration of adrenocortical suppression after dexamethasone administration is more variable in cats than in dogs. It appears that a dosage of 0.1 mg/kg of dexamethasone suppresses cortisol levels in healthy cats and cats with nonadrenal illness more reliably than 0.01 mg/kg of dexamethasone, which is the dosage used in dogs. Currently, the protocol most often used involves collection of blood samples for production of plasma or serum before and 4 and 8 hours after intravenous administration of 0.1 mg/kg of dexamethasone. Cortisol levels of less than 1 μg/dL at 4 and 8 hours are considered normal, levels between 1 and 1.4 μg/dL are borderline, and levels greater than 1.4 μg/dL are consistent with hyperadrenocorticism. Post-dexamethasone suppression of cortisol concentrations at 4 and 8 hours does not occur in cats with an FAT. Approximately 70% of cats with PDH show no or inadequate suppression.90
Measurement of Endogenous ACTH
Healthy cats may have very low ACTH concentrations, therefore the measurement of endogenous ACTH test can be used only after a diagnosis of hyperadrenocorticism has been made. Normal to elevated levels of ACTH support a diagnosis of PDH, and low to undetectable levels indicate an FAT. Sample handling is critical (see discussion earlier in this chapter).
High-Dose Dexamethasone Suppression Test
Compared with dogs, a higher dose of dexamethasone (1 mg/kg) has been advocated to differentiate between PDH and an FAT in cats; however, very limited information is available. Blood samples should be taken before and 4 and 8 hours after administration of dexamethasone. Suppression (less than 50% of baseline or less than 1.4 μg/dL) is consistent with PDH. No or insufficient suppression may be due either to an FAT or to PDH and cannot be used to differentiate between the two conditions.
Adrenal Ultrasonography
High-resolution transducers have made ultrasonographic visualization of the adrenal glands possible in cats, and reference ranges have been reported.92 Symmetric adrenal glands of normal or enlarged size are suggestive of PDH, whereas unilateral enlargement or an adrenal gland with a masslike appearance suggests an FAT. Detailed information on the ultrasonographic appearance of the adrenal glands in cats with hyperadrenocorticism currently is limited.
===========================================================================
UPDATE Date Added: 17 March 2005
Edward C. Feldman, DVM, DACVIM
High urinary corticoid/creatinine ratios in cats with hyperthyroidism.
Measurement of the urine cortisol: creatinine ratio (UCCR) provides an assessment of cortisol secretion over a period of time. This test is generally agreed to be an extremely sensitive screening test for the diagnosis of hyperadrenocorticism (HAC) in dogs and cats. The major concern regarding use of UCCR as the sole screening test in dogs has been its lack of specificity. In other words, dogs with naturally occurring HAC almost always have an abnormal UCCR, but dogs that have disorders other than HAC also commonly have abnormal test results. Therefore, while an abnormal UCCR test result can be used to support a clinical suspicion that a dog has HAC, it is generally recommended that a more specific test -- low dose dexamethasone suppression -- be used to "confirm" a diagnosis.
It was hypothesized in this clinical study that chronic excesses in thyroid hormone could influence the metabolic clearance of cortisol, thereby causing the UCCR to be abnormal in cats with naturally occurring hyperthyroidism. To evaluate the effect of thyroid hormone excess, UCCR results were measured in 32 cats with naturally occurring hyperthyroidism and in 45 healthy control cats. In 7 cats, the UCCRs were measured before and after treatment for hyperthyroidism. With data from the healthy cats, the reference range for the UCCR was determined to be 8.0 to 42.0 X 10-6. The UCCR results from the cats with hyperthyroidism were significantly higher than those of the control population. UCCR results from 15 of the 32 hyperthyroid cats were above the established reference range. Treatment of hyperthyroidism caused a marked decrease in UCCR in those 7 cats evaluated.
The results of this study demonstrate that UCCR results may be abnormally increased in cats with hyperthyroidism, probably because of increased metabolic clearance of cortisol and activation of the pituitary-adrenocortical axis by disease. Although the clinical features of hyperthyroidism and HAC in cats are different, the authors suggest that hyperthyroidism be ruled out when any cat is suspected of having HAC on the basis of an abnormal UCCR.
Results of this study also have larger implications. This study can be added to the list of reports that demonstrate lack of specificity associated with abnormal UCCR results in both cats and dogs. This lack of specificity strongly suggest that HAC never be confirmed in any dog or cat based only on an abnormal UCCR result. Rather, the diagnosis of HAC in both dogs and cats should be suspected after careful review of the history, physical examination, CBC, serum biochemical profile, urinalysis, and imaging studies. The diagnosis of HAC should be confirmed by other tests, such as the low dose dexamethasone test, in both dogs and cats.
de Lange, M.S., et al: High urinary corticoid/creatinine ratios in cats with hyperthyroidism. J Vet Int Med 2004;18:152-155.
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[求助]請問有人有經驗過使用干擾素治療FIP嗎?
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發表在 貓咪保健,
時間 2006-03-06 08:48 PM,
作者 changyuan
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我想我已經盡量在客觀, 我甚至可以引經據典告訴你是這麼回事, 引用這些資料...是我自己要花錢買的. 可是幫別人解決疑問並不需要你花一毛錢的.
從你的答案看不到任何感謝之意. 挖苦倒是很多!!!
我跟seanchen都有在查資料在給答案, 我不想再看到這種莫名奇妙的指控, 這也不值得我再為這種跟我無關的病例生氣, 你認為是就是, 但我同情你...至於同情你什麼...已經不再重要了....
就這樣.
ps:如果覺得我不適任, 請向壇主"用力"彈劾我, 我已經辭了三次, 壇主還是不讓我走...
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[求助]請問有人有經驗過使用干擾素治療FIP嗎?
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發表在 貓咪保健,
時間 2006-03-06 12:20 PM,
作者 changyuan
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為什麼說是無知?
類似文章po了多少, 看過了嗎?
爬文了嗎?
做kit就算確診嗎?
是獸醫無知嗎?
你的回覆很令人心寒, 枉費版工在這裡長期的po文....
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[推薦]不限資格之推薦獸醫區 ( 包括討論 )
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發表在 貓咪服務,
時間 2006-03-06 12:02 AM,
作者 changyuan
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來文若是無厘頭....必刪.
想直接建議的, 可以短訊通知版工.
特別是新手, 剛到人家家裡就嫌床小, 嫌客廳大, 嫌廁所太香...
是不是太沒禮貌了???
被刪文的新手....看到了沒??
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[討論]貓貓曾經有被檢查出梨型鞭毛蟲的請進
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發表在 貓咪保健,
時間 2006-03-05 11:53 PM,
作者 changyuan
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下面引用由晴天娃在 2006/03/05 04:25pm 發表的內容：
因為醫生說貓貓體內幾乎都有鞭毛蟲 不一定是水或環境問題
這個說法是不正確的
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[求助]請問有人有經驗過使用干擾素治療FIP嗎?
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發表在 貓咪保健,
時間 2006-03-05 11:45 PM,
作者 changyuan
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下面引用由VSLIN在 2006/03/05 10:23pm 發表的內容：
為啥版主可以強行貼文章~~真怪~~那選項等同沒用~
米國的FDA有准許使用INTERFERON~用在FIP~
那市米國的審查有問題摟~怪怪~
本人引述文章來自2005年新版的Ettinger內科, 而且是網路update版.
如該文所述, 嘗試用此法治療的獸醫所在多有. 然其效果為何...恕不再復述.
不過獸醫界這種學術鬥爭真的很令人厭煩, 別人不這麼做並不代表是庸醫, 而是有其他考量, 包括不想再讓動物受罪, 不想讓飼主花冤枉錢...等等.
如果有飼主願意嘗試當然可以如其所願, 但是這種治療不應該讓飼主懷抱太大期望才是.
另一方面, 從其他網友文章就可知很多病例動不動就被冠上此病, 真正被確診的病例又有幾?
飼主不懂是理所當然, 如果連所謂的專業獸醫也在亂搞, 實在是辜負本人當初一直提倡的社區家庭獸醫的觀念. 結果是很多病例送去教學醫院, 又被教學醫院的醫師悄悄轉往自己的診所去海撈一筆, 明明不是這種病說成是這種病, 最後就又塑造出神醫了, 倒楣的最後還是飼主.
我也不知道該不該同情這些無知的飼主....
誠如本人之前所言~~
我如果沒有辦法改變這個世界...我就會轉頭不去看它...
另...如果您認為您的文章有可讀性, 大可不必要人付費.
如果你的文章只是想封鎖版工, 老實告訴你....版工閱讀文章是沒有限制性的.
我所知道在美國很多的德高望重的獸醫, 都是很樂意與人分享與交流的.
您不妨打開心胸, 多跟大家交流吧.....
對不起,我不是在說敎....只是有感而發....敬請多多包涵...
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[求助]請問有人有經驗過使用干擾素治療FIP嗎?
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發表在 貓咪保健,
時間 2006-03-05 06:34 PM,
作者 changyuan
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下面引用由VSLIN在 2006/03/05 01:29pm 發表的內容：
使用interferon干擾素是要用α-2a喔~通常是人用的重組式干擾素~
現在米國有開始用貓重組式干擾素~據說效果還不錯~不過價格還是貴就是摟~
干擾素的作用是干擾病毒覆製~當初是發現感染兩種病毒會有互相牽制的效果~所以才叫它干擾素~干擾素的使用有兩種方式~高劑量的施打跟低劑量的口服~兩種方式取決於病的嚴重度~如果很嚴重就要用施打高劑量的方式~要控制就要低劑量口服來達到效果~
最近的治療研究方向是把用另一分子連結免疫複合體給白血球吃掉後~
避免血管通透度變大後產生腹水~有問題再問吧~
TREATMENT AND PREVENTION OF FELINE INFECTIOUS PERITONITIS
Virtually every cat with confirmed FIP dies. Some veterinarians prescribe immunomodulators (Propionobacterium acnes (immunoregulin), interferon, acemannan) to treat cats with FIP with no documented, controlled evidence of efficacy. Immunosuppressive drugs such as prednisone (4 mg/kg, orally once a day) or cyclophosphamide (2 to 4 mg/kg, orally four times a week) may slow disease progression but do not produce a cure. Cats with FIP should also be treated with broad-spectrum antibiotics and supportive therapy (subcutaneous fluids, rest, good nutrition, lack of stress) for as long as they are comfortable. Once disease signs become debilitating and weight and appetite decline, the owner must be prepared for the reality that their cat is dying.
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腎上腺素皮質機能亢進？
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發表在 貓咪保健,
時間 2006-03-05 09:32 AM,
作者 changyuan
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下面引用由gsw在 2006/03/04 11:39pm 發表的內容：
4.為什麼說和腎上腺素皮質亢進沒有關係呢？理由是什麼？
就是肝功能指數會飆很高.
特別是ALKP.
很奇怪,這種常識應該是每個獸醫都知道的...@@
測可體松需要臨床症狀符合, 血檢方面也吻合才做.
未臨床診治驟下斷語...還是要小心...不要隨便亂嚇人...
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腎上腺素皮質機能亢進？
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發表在 貓咪保健,
時間 2006-03-04 08:26 PM,
作者 changyuan
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下面引用由gsw在 2006/03/03 11:06pm 發表的內容：
ALKP:33u/l
ALT:16u/l
AST:79u/l
跟腎上腺素皮質機能亢進應該沒有關係.
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請問怪叔叔關於耳朵黴菌問題(已看過醫生)
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發表在 貓咪保健,
時間 2006-03-03 02:18 PM,
作者 changyuan
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下面引用由mi72914在 2006/03/03 03:10pm 發表的內容：
做過那些檢查 ( 包含抽血檢查的項目跟數據; 尿液分析值; 糞便鏡檢; 超音波; X光 ... ) 無
沒做檢查能做出診斷不是很奇怪嗎?
找個會做檢查的獸醫看看吧...
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[真正]貓咪論壇保健版所推薦的獸醫院.[永久徵稿中]
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發表在 貓咪保健,
時間 2006-03-02 07:17 PM,
作者 changyuan
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推 薦 人:BENNY
獸醫院名稱 : 小花動物醫院
獸醫院地址 : 新竹市新源街130號
獸醫院電話 : 03-5748460
推薦理由及個人經驗 :
我個人看診經驗:我較推薦看王院長，(另一個好像較專犬科)，前些時候，我的貓眼睛突然變一眼咪咪眼，帶去小花看，院長仔細用儀器先看眼鹼等等部份後，就拿出試劑和試紙分別測幾次確定病因後才跟我解釋，不是像部份醫生只有翻開看眼睛，就說病因，解釋得很粗淺
1.醫院內整潔，犬貓是分開診間，並非一般開放式的，診間內很乾淨，醫生看診仔細，儀器精密，診療細心詳細說明病情，有問必答
2.病房內有攝影機，能掌握住院動物的狀況
3.醫生也很有愛心與責任感，曾遇到有人拉不拉多(很小隻的幼犬)從樓下掉下來，好像不嚴重，主人就說要安樂，醫生不隨便答應
4.醫生對動物與飼主很有耐心，有時候我一遇到中途問題或是身邊貓狗的問題，我也會打電話先去問，醫生都會幫忙把疑問慢慢解答(提前是沒病人)
5.王院長自已也有養貓
6.關懷弱勢動物(遇在保護動物團體會盡力幫忙，也會有折扣)
7.院長是台灣少有的會做動物輪椅且有証書
以下文章就有提到
http://www.master-south.com.tw/modules/news/article.php?storyid=432
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小貓懷孕求助!!!
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發表在 貓咪保健,
時間 2006-02-23 09:35 PM,
作者 changyuan
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帶去看獸醫, 順便產檢吧.
由於壇主有交代
http://www.supervr.net/catbbs/topic.cgi?forum=11&topic=45&show=0
本文上鎖.
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